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		<title>Psy3241 - User contributions [en]</title>
		<link>http://72.14.177.54/psy3241/Special:Contributions/Kgutekunst</link>
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		<item>
			<title>Pascual-Leone et al. (1995)</title>
			<link>http://72.14.177.54/psy3241/Pascual-Leone_et_al._(1995)</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Plasticity Symposium]]&lt;br /&gt;
The Role of Reading Activity on the Modulation of Motor Cortical Outputs to the Reading Hand in Braille Readers&lt;br /&gt;
&lt;br /&gt;
Discusses the plasticity of the brain with skilled activities. In this study, braille readers had changes in their brain imaging through TMS (Transcranial Magnetic Stimulation) and cortical output maps showed differences on days when participants were reading braille. &lt;br /&gt;
Generally, the study suggests that the brain changes with regard to what activities a person is doing, with the idea that the brain accounts for the fact that people will not be doing the same things with their hands, or with their legs, all the time. Furthermore, plastic changes in the CNS are required for learning a new skill. The proficiency of a skill may be dependent on the capacity of modulating these intracortical connections rapidly when necessary. Similarly, modulation of motor cortical outputs can be reverted with lack of practice.&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:49:33 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Pascual-Leone_et_al._(1995)</comments>		</item>
		<item>
			<title>Witthoft and Winawer (2006)</title>
			<link>http://72.14.177.54/psy3241/Witthoft_and_Winawer_(2006)</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Discussion */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Synesthesia Symposium]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Synesthetic Colors Detirmined by Having Colored Refrigerator Magnets in Childhood==&lt;br /&gt;
Presentation by: Mandy French&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Introduction ==&lt;br /&gt;
&lt;br /&gt;
•	Inducer- stimulus that produces synesthesia&lt;br /&gt;
&lt;br /&gt;
•	Concurrent- the synesthesia itself&lt;br /&gt;
&lt;br /&gt;
•       The relationship between inducer and concurrent arises from pre-existing mappings between sensory areas that are overactive or fail to be pruned during development.  &lt;br /&gt;
&lt;br /&gt;
•	One theory suggests that all infants are innately synesthetic with sensory differentiation only coming with development and the pruning of connections. Another theory indicates that synesthesia is learned through particular inducer and concurrent pairings from sensory information in the environment.  &lt;br /&gt;
&lt;br /&gt;
•	Word-taste synesthesia involves the semantics and phonology of speech influence the relationship between inducer and concurrent. The phoneme K tends to elicit the taste of foods with the same phoneme (cake).&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Main Study ==&lt;br /&gt;
&lt;br /&gt;
•	The participant AED has color grapheme synesthesia where the letters of the alphabet had colors learned from refrigerator magnets.  &lt;br /&gt;
&lt;br /&gt;
•	Moved to Russia at age 3, so synesthesia transferred to the Russian alphabet, Cyrillic.&lt;br /&gt;
&lt;br /&gt;
•	Reports all achromatic, or non-color, text as having colors overlaid on the surfaces of letter or numbers.&lt;br /&gt;
&lt;br /&gt;
•	Transfer from English to Cyrillic letters showed consistency in color when there were phonemic or visual similarities. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Methods ==&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
•	Testing was done on 2 separate occasions, 21 days apart.&lt;br /&gt;
&lt;br /&gt;
•	Graphemes appeared in Grey and AED adjusted the hue, brightness and saturation by using controls on the screen. There were 360 possible hues, 100 levels of saturation and 128 levels of brightness.&lt;br /&gt;
&lt;br /&gt;
•	Consistency was measured by individually correlating the hue, brightness and saturation of each letter.&lt;br /&gt;
&lt;br /&gt;
•	Each matching session (unlimited time) consisted of the digits 0-9 and all letters of the alphabet, both upper and lowercase, were presented in random order&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Results==&lt;br /&gt;
&lt;br /&gt;
'''EXPERIMENT 1- CONSISTANCY OVER TIME'''&lt;br /&gt;
&lt;br /&gt;
•	AED completed task in 15 minutes on each test&lt;br /&gt;
&lt;br /&gt;
•	Fulfills criteria for this measure of synesthesia&lt;br /&gt;
&lt;br /&gt;
•	Hues of individual letter from both tests were highly correlated&lt;br /&gt;
&lt;br /&gt;
•	Saturation and brightness were more variable&lt;br /&gt;
&lt;br /&gt;
'''EXPERIMENT 2- TRANSFER TO CYRILLIC'''&lt;br /&gt;
&lt;br /&gt;
•	Significant correlations for hue and saturation but not brightness were found&lt;br /&gt;
&lt;br /&gt;
•	Near perfect correlation for hue with Cyrillic letters that have phonetic similarity to English letters but not for saturation or brightness&lt;br /&gt;
&lt;br /&gt;
'''EXPERIMENT 3- LEVEL OF REPRESENTATION OF THE INDUCER''' &lt;br /&gt;
&lt;br /&gt;
•	Importance of visual similarity of upper to lowercase letters on saturation and brightness was looked at&lt;br /&gt;
&lt;br /&gt;
•	No significant effects for brightness were found &lt;br /&gt;
&lt;br /&gt;
•	Main effect for case and font in English letters; uppercase letters were more saturated than lowercase letters and times new roman was more saturated than cursive.&lt;br /&gt;
&lt;br /&gt;
•	The main effect for Cyrillic letters were much greater in uppercase letters than lowercase letters, and only for the Cyrillic letters that are visually similar to the English counterparts.&lt;br /&gt;
&lt;br /&gt;
•	The times new roman font led to more saturation than the cursive font. This could be due to the fact that the refrigerator magnets were more similar to times new roman.&lt;br /&gt;
&lt;br /&gt;
'''EXPERIEMNT 4- LEVEL OF REPRESENTATION IN THE CONCURRENT'''&lt;br /&gt;
&lt;br /&gt;
•	Examine if the concurrent synesthesia is affected by a lightness constancy illusion. &lt;br /&gt;
&lt;br /&gt;
•	Achromatic letters appear embedded in one of the two lightness illusions checker-shadow or snakes illusion.&lt;br /&gt;
&lt;br /&gt;
•	AED showed a highly significant effect in both the checker-shadow and snakes illusions, which demonstrates that her concurrent synesthesia occurs before computing lightness in visual processing.&lt;br /&gt;
&lt;br /&gt;
== Discussion ==&lt;br /&gt;
•	DNA may be a predictor for synesthesia, but it does not determine the acquisition of synesthesia. As Withoft and Winawer (2006) display in their experiment, synesthesia can be learned from early childhood experiences with environmental stimuli such as colored refrigerator magnets.&lt;br /&gt;
&lt;br /&gt;
See also: [[Synesthesia]]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:44:30 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Witthoft_and_Winawer_(2006)</comments>		</item>
		<item>
			<title>Speling et al. (2006)</title>
			<link>http://72.14.177.54/psy3241/Speling_et_al._(2006)</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Synesthesia Symposium]]&lt;br /&gt;
&lt;br /&gt;
See also: [[Synesthesia]]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:44:04 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Speling_et_al._(2006)</comments>		</item>
		<item>
			<title>Nikolic et al. (2007)</title>
			<link>http://72.14.177.54/psy3241/Nikolic_et_al._(2007)</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Conclusion */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Synesthesia Symposium]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
Color Opponency in Synaesthetic Experiences&lt;br /&gt;
by Danko Nikolic, Philipp Lichti, and Wolf Singer&lt;br /&gt;
&lt;br /&gt;
== Synaesthesia? ==&lt;br /&gt;
&lt;br /&gt;
Synaesthesia is a harmless perceptual condition in which there is a blending of the senses. Generally letters and numbers are seen as different colors or where music initiates the perception of color. A synaesthetic individual could be said to have the ability to see sound. Some synaesthetes are categorized as associators which is the perception of a color on an internal screen or in the mind, while others are considered projectors where color is seen on objects in space such as colored letters on a page.&lt;br /&gt;
&lt;br /&gt;
==The Stroop Test ==&lt;br /&gt;
&lt;br /&gt;
A Stroop test which was developed in 1935 by J. R. Stroop, an individual is instructed to name the ink color of a word that refers to a color. The reaction times to identify the ink color for the individual is measured and analyzed. Reaction times are generally longer if the color of the ink and the meaning of the word do not match (incongruent condition), for example if yellow is written in red ink. This test can be applied to study synaesthetic individuals by adjusting the meaning of the words to match the perceptual experience a person has to that word. In summary, a synaesthetic person would look at a word that evokes a red perception that would be colored in red ink and would be able to react faster than a non-synaesthetic individual.&lt;br /&gt;
&lt;br /&gt;
== Color Opponency in Synaesthetic Experiences ==&lt;br /&gt;
&lt;br /&gt;
In the current study researchers are taking advantage of the color opponent receptive fields of the brain. The color opponent receptive fields are cells that are excited by red and inhibited by green and cells that are excited by yellow and inhibited by blue. This fact allows researchers to study which receptive fields are active during real and synaesthetic perceptions.&lt;br /&gt;
&lt;br /&gt;
== Experiment 1 ==&lt;br /&gt;
&lt;br /&gt;
===Subjects===&lt;br /&gt;
6 synaesthetic individuals participated in the study, four were women and two were men and five of these people had other forms of Synaesthesia. 12 nonsynaesthetes participated in the study as the control group who matched the synaesthetes in gender and age.&lt;br /&gt;
&lt;br /&gt;
===Procedure===&lt;br /&gt;
The subjects were tested on color associations. There were three conditions: the congruent condition in which the color of each grapheme was the same as the synaesthetic color; the incongruent opponent condition in which the color of each grapheme was opposite to the synaesthetic color; the incongruent independent condition in which the color of each grapheme and the synaesthetic color were represented by different opponent-color channels; and the baseline condition in which the experimenters used a grapheme that did not have a synaesthetic color association. The experiment took place in a dimly lit room with a computer running the visual stimulation tool. The subjects were given 200 trials in which they were told to accurately name the real color of each grapheme as fast and as they could. The entire experiment took approximately 25 minutes. &lt;br /&gt;
&lt;br /&gt;
===Results===&lt;br /&gt;
The subjects response accuracy was very high (98%) but a Tukey HSD test for a post hoc comparison indicated that subjects named the correct color faster in the congruent condition than in the incongruent condition. The experimenters found significant results between the incongruent independent condition (which was named faster) and the incongruent opponent condition. They found that opponent incongruent colors produced more interference than the independent incongruent colors. They also found that the congruent synaesthetic colors helped the subjects to name the real colors of the graphemes. &lt;br /&gt;
&lt;br /&gt;
==Experiment 2==&lt;br /&gt;
The experimenters examined semantic associations between shape and color using the Stroop task. The stimuli that they used were commonly known everyday associations between shape and color. The experimenters hypothesized that semantic associations do not involve the opponent-color system. They used four of the synaesthetes and 8 of the control subjects from experiment 1. They also used the same methods as for the synaesthetic Stroop test except that only three objects were used and each only appeared in three stimulation conditions. The subjects were presented each stimulus 25 times, given a total of 225 trials overall. &lt;br /&gt;
&lt;br /&gt;
===Results===&lt;br /&gt;
Once again, the response accuracy was very high, indicating that color opponency does in fact affect the semantic associations between shape and color differently than synaesthetic associations. &lt;br /&gt;
&lt;br /&gt;
==Conclusion==&lt;br /&gt;
&lt;br /&gt;
Overall, the experimenters concluded that opponent synaesthetic and real colors interfere the most with the naming and perception of a real color. Conversely, when synaesthetic and real colors are identical, the color-naming process is assisted and the response times are decreased. These findings show that the color experiences stimulated by this experiment involve color-opponent channels and thus neurons in the V1 to V4/V8 areas. The results of experiments 1 and 2 suggest that the semantic associations between graphemes and colors explain the interference between nonopponent colors.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
See also: [[Synesthesia]]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:43:44 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Nikolic_et_al._(2007)</comments>		</item>
		<item>
			<title>Kim et al. (2006)</title>
			<link>http://72.14.177.54/psy3241/Kim_et_al._(2006)</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Synesthesia Symposium]]&lt;br /&gt;
'''Perceptual Interaction Between Real And Synesthetic Colors'''&lt;br /&gt;
In this article the perceptual interaction between real and synesthetic colors is examined using participants with color-graphemic synesthesia. People with color-graphemic synesthesia experience consistent and vivid colors after viewing achromatic alphanumeric characters. This article looks at the issues of how real and synesthetic colors interact, and how synesthetes see the real and concurrent synesthetic color at the same time. Other issues include how one can see two colors in the same location, and what the relationship is between synesthesia and imagery. &lt;br /&gt;
&lt;br /&gt;
In the first experiment, the influence that real and synesthetic colors play on the perceived direction of apparent motion was examined. In the interaction condition, the first frame was shown which was non-inducing, and physically colored. SHortly after a second frame was shown which was achromatic with different characters. The results showed that synesthetes were experiencing the illusion of motion as the first frame had real red objects in it and the second frame had synesthetically red colored objects which gave them the illusion of motion they were seeing.&lt;br /&gt;
&lt;br /&gt;
In the second experiment, the interaction between real and synesthetic colors that results in binocular rivalry was examined. Binocular rivalry is when two different images compete for perceptual dominance. In this experiment they were looking for the rivalry between real and synesthetic colors. In the interaction condition both LR and WO perceived the real color and synesthetic color during binocular rivalry. This observation of the grouping of real and synesthetic colors together was comparable to the grouping between synesthetic colors and the grouping of actual colors. &lt;br /&gt;
&lt;br /&gt;
The results show that their is  a significant interaction between the real and synesthetic colors during perceptual grouping. Also, the interaction between real and synesthetic colors result in a perception in specific motion and character grouping. [http://blackboard.rollins.edu/courses/1/10301.PSY324.1.200801/content/_176453_1/Kim_2006_interaction_real_synesthetic_color.pdf Link to article]&lt;br /&gt;
&lt;br /&gt;
See also: [[Synesthesia]]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:43:21 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Kim_et_al._(2006)</comments>		</item>
		<item>
			<title>Hancock et al. (2006)</title>
			<link>http://72.14.177.54/psy3241/Hancock_et_al._(2006)</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Questions */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Synesthesia Symposium]]&lt;br /&gt;
'''Monozygotic Twins' Colour-Number Association: A Case Study'''&lt;br /&gt;
   Presentation By Mandy French&lt;br /&gt;
&lt;br /&gt;
== Introduction ==&lt;br /&gt;
•	Investigates the color-number associations of monozygotic twins age 12 at testing&lt;br /&gt;
&lt;br /&gt;
•	Origin of color-number association is known to be a colored number jigsaw puzzle&lt;br /&gt;
 &lt;br /&gt;
•	At age 3 the association was first noted when a teacher asked the boys to report numbers, but they insisted on reporting color names&lt;br /&gt;
&lt;br /&gt;
•	The colors that were reported were synonymous with the colors shown on the “Early Learning Centre” number jigsaw puzzle that the boys regularly played with&lt;br /&gt;
&lt;br /&gt;
•	The brothers showed the same color to number pairings, but the association was not quit as strong for one of the brothers&lt;br /&gt;
&lt;br /&gt;
•	By age 12 the boys report what color a digit is as well as most letters&lt;br /&gt;
&lt;br /&gt;
•	Researcher was interested in determining if the boys show the Stroop-like interference effects that are commonly used and if they had enduring associations&lt;br /&gt;
&lt;br /&gt;
== Methods ==&lt;br /&gt;
•	Name the color in which a digit is displayed&lt;br /&gt;
&lt;br /&gt;
•	Hypothesized that if the digit is colored according to the jigsaw puzzle, then the boys will be quicker when compared to incorrectly colored digits&lt;br /&gt;
&lt;br /&gt;
== Participants ==&lt;br /&gt;
•	Monozygotic twin boys aged 12 years and 2 months&lt;br /&gt;
&lt;br /&gt;
•	Physically and intellectually similar&lt;br /&gt;
&lt;br /&gt;
== Materials ==&lt;br /&gt;
•	The twins separately selected colors for each of the digits from 0-9&lt;br /&gt;
&lt;br /&gt;
•	Numbers were displayed in 200pt font about 6cm high on the screen&lt;br /&gt;
&lt;br /&gt;
•	The color selections the boys made were stored as congruent colors&lt;br /&gt;
&lt;br /&gt;
•	Incongruent versions were created by changing the colors of discordant pairs&lt;br /&gt;
&lt;br /&gt;
•	The swaps were all for adjacent numbers&lt;br /&gt;
&lt;br /&gt;
== Procedure ==&lt;br /&gt;
•	Asked to name the color of the displayed digit as rapidly as possible&lt;br /&gt;
&lt;br /&gt;
•	Each digit was presented 8 times in one run of the experiment&lt;br /&gt;
&lt;br /&gt;
•	Each digit was presented 4 times congruent and 4 times incongruent per run&lt;br /&gt;
&lt;br /&gt;
•	Each boy ran the procedure 6 times over a couple of days&lt;br /&gt;
&lt;br /&gt;
•	A total of 24 trials were conducted for each digit&lt;br /&gt;
&lt;br /&gt;
== Results ==&lt;br /&gt;
•	49 of the 480 trials were removed for R and 51 were removed for T because the voice trigger failed to detect the word or was triggered by an extraneous noise&lt;br /&gt;
&lt;br /&gt;
•	38 trials for R and 19 trials for T were removed because they were more than 2 standard deviations away from the mean&lt;br /&gt;
&lt;br /&gt;
•	Both boys had an increased reaction time to incongruent colors&lt;br /&gt;
&lt;br /&gt;
•	An Anova demonstrates an effect of congruence, but not a significant difference between the boys&lt;br /&gt;
&lt;br /&gt;
•	R made more color naming errors than T&lt;br /&gt;
&lt;br /&gt;
•	11 errors for R and 1 error for T&lt;br /&gt;
&lt;br /&gt;
•	It is believed that T was more careful, which resulted in his lower error rate and slower reaction time&lt;br /&gt;
&lt;br /&gt;
== Discussion ==&lt;br /&gt;
•	“Associator” Synaesthetes can see colors in the “mind’s eye”&lt;br /&gt;
&lt;br /&gt;
•	“Projector” Synaesthetes see the color as an overlay on the digit&lt;br /&gt;
&lt;br /&gt;
•	The boys experience is consistent with the finding that they do not report photisms or any sense of perceiving color, they just know that a particular number is a specific color&lt;br /&gt;
&lt;br /&gt;
•	Elias et al. showed the synaesthete showed much stronger activation in visual areas on several tasks than the learned associator&lt;br /&gt;
&lt;br /&gt;
•	It would be interesting to use fMRI to see in which group of synaesthetes the boys fit into &lt;br /&gt;
&lt;br /&gt;
•	The boy’s association is learned, but not practiced&lt;br /&gt;
&lt;br /&gt;
•	Both twins report the color-number association and do so with a very similar set of colors&lt;br /&gt;
&lt;br /&gt;
•	Even though both boys had not seen the jigsaw puzzle for at least 7 years at the time of testing, the accuracy of the match was remarkable&lt;br /&gt;
&lt;br /&gt;
•	It is estimated that the frequency of synaesthesia is 1 in 25,000&lt;br /&gt;
&lt;br /&gt;
•	Recent study demonstrates the possibility of the rate of synaesthesia being as high as 1 in 110&lt;br /&gt;
&lt;br /&gt;
•	Synaesthesia may not be an all or nothing effect, but may be more of a graded phenomenon with enduring associations at one end of the scale and full blown cross modal perception at the other&lt;br /&gt;
&lt;br /&gt;
•	R and T seem to fit at the mild end of the range&lt;br /&gt;
&lt;br /&gt;
•	R and T have reliable, long lasting associations that cause a Stroop interference, even though they do not report seeing anything&lt;br /&gt;
&lt;br /&gt;
•	A genetic effect for synaesthesia is possible because the boys’ mother also displayed a color-number association&lt;br /&gt;
&lt;br /&gt;
== Questions ==&lt;br /&gt;
Is it possible that if the twins practiced the learned association that the color-number association could grow stronger?&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
See also: [[Synesthesia]]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:42:52 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Hancock_et_al._(2006)</comments>		</item>
		<item>
			<title>Aleman et al. (2001)</title>
			<link>http://72.14.177.54/psy3241/Aleman_et_al._(2001)</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Importance */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Synesthesia Symposium]]&lt;br /&gt;
&lt;br /&gt;
Activation of the Striate Cortex in the absence of visual stimulation: an fMRI Study of Synesthesia- Presented by Hayley Devlin and Rachel Kimchi&lt;br /&gt;
&lt;br /&gt;
Shadowed by Lauren Malonson and Meredith Walsh&lt;br /&gt;
&lt;br /&gt;
=Key Terms=&lt;br /&gt;
&lt;br /&gt;
'''Synesthesia-'''&lt;br /&gt;
&lt;br /&gt;
This occurs when one sensory system in your body triggers a perceptual experience in another unrelated system. An common example of this is when a person hears a sound then will immediately see a color or shape in the mind's eye as a response to the sound. People who have synethesia are referred to as synethetes and are often unaware that there is anything wrong with them because it develops at such a young age. This is also the reason why researchers have a vague idea about how many people have synesthesia. &lt;br /&gt;
&lt;br /&gt;
'''Primary Visual Cortex, Striate Cortex, V1'''&lt;br /&gt;
&lt;br /&gt;
Located in the brain's occipital lobe, the primary visual cortex is part of the neocortex that receives visual input from the receptive cells of the retina.&lt;br /&gt;
&lt;br /&gt;
                                                 http://www.aph.org/cvi/images/brain_2.jpg&lt;br /&gt;
&lt;br /&gt;
=Overview of Article=&lt;br /&gt;
&lt;br /&gt;
The purpose of this study was to find out if the primary visual cortex is activated without direct external input to the visual system.  In other words, the question is, does the primary visual cortex play a functional role in internally generated visual perception?  To determine this, an fMRI scan was conducted on a woman with color-word synesthesia (when she heard a word, she involuntarily saw a color) during two verbal tasks and control conditions.  In the first task (passive listening), the participant was auditorily presented with 14 words, and in the next (verbal fluency), she was presented with one letter at a time and asked to generate as many words as possible starting with that letter.  In the control conditions, tones were presented and the participant pressed a button when the frequency changed.  All fMRI results were compared against those of normal participants.&lt;br /&gt;
&lt;br /&gt;
=Results=&lt;br /&gt;
&lt;br /&gt;
Results showed that V1 was activated in both the passive listening and the verbal fluency tasks in the participant with color-word synesthesia. The normal subjects did not show activation of V1 in any condition.  &lt;br /&gt;
&lt;br /&gt;
=Importance=&lt;br /&gt;
&lt;br /&gt;
These results are significant for several reasons:  first, while previous studies have tested the role of V1 in the presence of visual perception (in the absence of visual stimulation), those studies had mixed results;  additionally, the previous studies focused on conscious visual perception, whereas this one focused on involuntarily generated perception;  next, these results, as suggested in the study, may have important implications regarding other types of visualization that are uncontrolled by the experiencer (such as hallucinations);  and finally, the results provide significant evidence that feedback connections into V1 may &amp;quot;mediate processes such as perceptual organization, attention and awareness,&amp;quot; and that those connections play a major role in &amp;quot;associative and primary visual areas in visual experience&amp;quot; in the absence of direct visual stimulation.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
See also: [[Synesthesia]]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:42:10 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Aleman_et_al._(2001)</comments>		</item>
		<item>
			<title>Palmeri et al. (2002)</title>
			<link>http://72.14.177.54/psy3241/Palmeri_et_al._(2002)</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Synesthesia Symposium]]&lt;br /&gt;
&lt;br /&gt;
Introduction to WO &lt;br /&gt;
WO has lexical synesthesia, which means that achromatic words and alphanumeric characters are seen in bright and consistent colors. The subject, WO has experienced this form of synesthesia since childhood and describes that he experiences a visual imagery synesthesia in which spoken words evoke consistent colors in “his mind’s eye” (Pamleri et al., 2000, p.4127). Despite his synesthesia, he has normal trichromatic color vision, good stereopsis and normal visual acuity. &lt;br /&gt;
&lt;br /&gt;
Method and Results&lt;br /&gt;
The authors tested WO’s synesthesia as elicited by both local and global forms. (i.e. a large number “5” made of small “2s”) Depending on what the subject was attending to, the local or global form of the stimulus, WO experienced whatever color was consistently associated with that stimulus (i.e. light green for the “5” and orange for the “2s”). WO also elicited synesthesia in response to stereopic displays resulting from binocularly defined stimuli. The structure-from-motion defined stimuli, which consisted of arrows facing in different directions, which formed the percept of a number. WO readily demonstrated synesthesia by identifying the color for the number displayed. Palmeri et al. (2000) also conducted a modified stroop interference task in which WO was asked to name colors either congruent or incongruent with the ink color, using latency as the independent variable. WO showed significantly slower reaction time when the written color was incongruent with his synesthesia. In another modified stroop task WO was asked to name the color of the word, which was either congruent or incongruent with the colors normally evoked by his synesthetic experience. The reaction times evoked to name words printed in grey (control condition) had similar durations to the words printed in the appropriate synesthetic color. The final task included a visual search efficiency test consisting of comparable visual stimuli (i.e. a “2” among numerous “5s” or a “6” among numerous “8s”). The results showed that WO’s error rates and reaction times were significantly lower and than that of controls and he commented that the target number was easily distinguishable compared to the other digits. &lt;br /&gt;
&lt;br /&gt;
Discussion&lt;br /&gt;
&lt;br /&gt;
Synesthesia is an automatic condition that results in multimodal perceptual experiences from a unimodal sensory stimulus. Palmeri et. al. (2000) reported, “it appears that the binding of color and form takes place during the process of form recognition itself, with synesthetic colors available before the explicit recognition of the digits (p. 4130). The authors conclude that lexical synesthesia binds color to form before the synesthete consciously recognizes the stimulus. Thus, it seems that synesthesia occurs during central visual processing, instead during higher order, semantic processing.&lt;br /&gt;
&lt;br /&gt;
See also: [[Synesthesia]]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:26:48 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Palmeri_et_al._(2002)</comments>		</item>
		<item>
			<title>File:Amygdala and Autism.JPG</title>
			<link>http://72.14.177.54/psy3241/File:Amygdala_and_Autism.JPG</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:22:35 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/File_talk:Amygdala_and_Autism.JPG</comments>		</item>
		<item>
			<title>Schizophrenia</title>
			<link>http://72.14.177.54/psy3241/Schizophrenia</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Treatments */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological syndromes]]&lt;br /&gt;
Schizophrenia (from the Greek roots schizein &amp;quot;to split&amp;quot; and phrēn &amp;quot;mind&amp;quot;) is a chronic, severe, and disabling brain disorder that affects almost 2 percent of the U.S. population in their lifetime. This mental illness is characterized by impairments in perception that include hallucinations, delusions, and disorganized behavior. Unlike some speculations, schizophrenia does not mean &amp;quot;split personality&amp;quot;. Schizophrenia affects both men and women, men usually earlier than women, anywhere from the late teens to early thirties. Rare cases of schizophrenia have also been reported in children.&lt;br /&gt;
&lt;br /&gt;
At the beginning, schizophrenia was characterized in three ways which were disorganized, catatonic and paranoid. Now there have grown to be 5 sub-classifications that go into diagnosing schizophrenia which include: disorganized (hebephrenic), catatonic, paranoid, residual, and undifferentiated. &lt;br /&gt;
&lt;br /&gt;
There are many areas of the brain that are affected when schizophrenia is detected. The basal ganglia which is involved in movement, emotions, and sensory information, can lead to hallucinations and paranoia. In the frontal lobe that controls problem-solving and critical thinking, difficutly in planning and organizing thoughts may occur. The limbic system that is involved with emotions can contribute to agitation. The learning and memory part of the brain, or the hippocampus, can be affected. The occipital lobe, or the visual part of our brain, can also lead to hallucinations. Also, the auditory system that allows us to understand speech, can create auditory hallucinations. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Symptoms &amp;amp; Signs ==&lt;br /&gt;
There are positve (productive) and negative (deficit) symptoms of schizophrenia. Positive symptoms include the following:&lt;br /&gt;
psychotic episodes, which is a displacement from what is real and unreal; delusions, or false judgements or beliefs; hallucinations, which are strong subjective perceptions of an object or event which is non-existent that may affect any or all sensory perceptions; disorganized speech or behavior, and thought disorder or cognitive dysfuntion. Negative symptoms on the other hand include: social and occupational dysfunction, lack of motivation, withdrawl, and loss of concentration, loss of emotional tone or reaction, and the inability to articulate. For schizophrenia to be taken into consideration, symptoms should be evident for about 6 months. Eugen Bleuler, who originated the term schizophrenia, referred to it as a split between subjective feeling, or affect, and the thought being experienced.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Treatments ==&lt;br /&gt;
Although, schizophrenia has been seen to have genetic factors, there is still no determination of what causes this disease. However, there are many medications, such as antipsychotic medication, that help reduce the symptoms so schizophrenic people can live a happy and independent life. Along with medication, there is also what is called supplemental therapies which include psychosocial or cognitive therapy, rehabilitation day programs, peer support groups, and nutritional supplements that can help. More intense therapies are electroconvulsive therapy and transcranial magnetic stimulation (TMS). A healthy diet and even amino acids and antioxidant vitamins such as glycine or sarcosine supplements can be used at treatments.&lt;br /&gt;
&lt;br /&gt;
Video of Schizophrenia drugs: [http://www.youtube.com/watch?v=3V1hXhX26Gw]&lt;br /&gt;
&lt;br /&gt;
== History ==&lt;br /&gt;
&lt;br /&gt;
The term schizophrenia is a relatively new word that originated less than 100 years ago, however, the disease has been said to have been around for all mankind. Schizophrenia was first identified as an actual mental illness by Dr. Emile Kraepelin in 1887 but he described the disease as &amp;quot;dementia praecox&amp;quot;. It wasn't until 1911 that a Swiss psychiatrist named Eugen Bleuler named the actual disease as schizophrenia. Since then, the definition of schizophrenia continues to change as we learn more about this mental illness.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
[http://www.schizophrenia.com/]&lt;br /&gt;
[http://www.medicinenet.com/schizophrenia/article.htm]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:19:20 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Schizophrenia</comments>		</item>
		<item>
			<title>Broca's area</title>
			<link>http://72.14.177.54/psy3241/Broca%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Description */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
'''Broca's area''' is the area in the brain that is responsible for speech production, language processing, and language comprehension. Broca's area was first discovered in 1861 by Pierre [[Paul Broca]], after studying the postmortem brain of Monsieur LeBorgne, commonly known as Tan, a patient of his who had a speech impediment.&lt;br /&gt;
&lt;br /&gt;
== Description ==&lt;br /&gt;
Broca's area is located in the left frontal lobe, around the opercular and triangular sections of the inferior frontal gyrus. It is connected to Wernicke’s area of the brain by the arcuate fasciculus, which is a pathway made of neurons. (see below).&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
[[Image:Broca.jpg]]&lt;br /&gt;
&lt;br /&gt;
== Parts ==&lt;br /&gt;
Broca's area contains two main parts: the ''Pars triangularis'' and the ''Pars opercularis''.&lt;br /&gt;
&lt;br /&gt;
The '''Pars triangularis''' is located in the anterior part of Broca's area. Researchers believe that this area of the brain is responsible for helping the human brain interpret different stimulus modes. It also supports the programming of verbal conducts.&lt;br /&gt;
&lt;br /&gt;
The '''Pars opercularis''' is located in the posterior part of Broca's area. It is believed that this area supports only one stimulus mode, rather than multiple modes like the Pars triangularis. This portion of Broca’s area is also believed to coordinate the organs used for speech in order to produce language.&lt;br /&gt;
&lt;br /&gt;
== Damage to Broca's area ==&lt;br /&gt;
If Broca's area is damaged, people will usually suffer from a condition called ''[[Broca's aphasia]]''. This condition is also sometimes called ''expressive aphasia'', ''nonfluent aphasia'', or ''motor aphasia''. Broca's aphasia makes people unable to create sentences that are grammatically complex. In addition, the sentences usually contain very few words related to content. Broca's aphasia is characterized by nonfluent speech, few words, short sentences, and many pauses.&lt;br /&gt;
&lt;br /&gt;
For example, if a Broca's aphasic was trying to explain how he came to the hospital for dental surgery, it might sound like this:&lt;br /&gt;
&lt;br /&gt;
&amp;quot;Yes... ah... Monday... er... Dad and Peter H... (his own name), and Dad.... er... hospital... and ah... Wednesday... Wednesday, nine o'clock... and oh... Thursday... ten o'clock, ah doctors... two... an' doctors... and er... teeth... yah.&amp;quot;&lt;br /&gt;
&lt;br /&gt;
Despite the Broca's aphasic's difficulty in putting together sentences, a person with a damaged Broca's area is generally capable of comprehending language without a problem. Sometimes, however, the person may have difficulty with understanding a few words used in a sentence with complex syntax. These people typically have damage only in the posterior part of Broca's area, a condition called ''[[Wernicke’s aphasia]]''. Those suffering from Wernicke’s aphasia may have somewhat normal speech, though it tends to be vague or even meaningless.&lt;br /&gt;
&lt;br /&gt;
== Video Clip ==&lt;br /&gt;
[http://www.youtube.com/watch?v=iuPeOKeCxu8 Broca's area video]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:08:53 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Broca%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[Image:wernickesarea.gif]]&lt;br /&gt;
&lt;br /&gt;
Wernicke's area was discovered by [[Carl Wernicke]] in 1874. Located on the left superior temporal gyrus, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to [[Broca's area]]. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''[[Wernicke's aphasia]]''. Recent research by Dronkers et. al. (1998) revealed that 'pure' damage to Wernicke's area most likely results in impairment in repetition rather than comprehension deficits. Damage to the part of the brain linking Broca's area and Wernicke's area, the arcuate fasciculus, can lead to conduction aphasia, in which the patient loses the ability to repeat words.&lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: [[Broca's area]], the supramarginal gyrus, [[angular gyrus]], and the arcuate fasciculus (a pathway thought to connect Wernicke's area with Broca's area).&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Stirling, J. (2002). Introducing neuropsychology. New York: Psychology Press.&lt;br /&gt;
&lt;br /&gt;
Ogden, J. A. (2005). Fractured minds. New York: Oxford University Press. &lt;br /&gt;
&lt;br /&gt;
Image taken from: &lt;br /&gt;
http://users.fmrib.ox.ac.uk/~stuart/thesis/chapter_3/section3_2.html&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:07:36 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[Image:wernickesarea.gif]]&lt;br /&gt;
&lt;br /&gt;
Wernicke's area was discovered by [[Carl Wernicke]] in 1874. Located on the left superior temporal gyrus, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to [[Broca's area]]. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. Recent research by Dronkers et. al. (1998) revealed that 'pure' damage to Wernicke's area most likely results in impairment in repetition rather than comprehension deficits. Damage to the part of the brain linking Broca's area and Wernicke's area, the arcuate fasciculus, can lead to conduction aphasia, in which the patient loses the ability to repeat words.&lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: [[Broca's area]], the supramarginal gyrus, [[angular gyrus]], and the arcuate fasciculus (a pathway thought to connect Wernicke's area with Broca's area).&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Stirling, J. (2002). Introducing neuropsychology. New York: Psychology Press.&lt;br /&gt;
&lt;br /&gt;
Ogden, J. A. (2005). Fractured minds. New York: Oxford University Press. &lt;br /&gt;
&lt;br /&gt;
Image taken from: &lt;br /&gt;
http://users.fmrib.ox.ac.uk/~stuart/thesis/chapter_3/section3_2.html&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:06:59 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Broca's area</title>
			<link>http://72.14.177.54/psy3241/Broca%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Description */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
'''Broca's area''' is the area in the brain that is responsible for speech production, language processing, and language comprehension. Broca's area was first discovered in 1861 by Pierre [[Paul Broca]], after studying the postmortem brain of Monsieur LeBorgne, commonly known as Tan, a patient of his who had a speech impediment.&lt;br /&gt;
&lt;br /&gt;
== Description ==&lt;br /&gt;
Broca's area is located in the left frontal lobe, around the opercular and triangular sections of the inferior frontal gyrus. It is connected to [[Wernicke’s area]] of the brain by the arcuate fasciculus, which is a pathway made of neurons. (see below).&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
[[Image:Broca.jpg]]&lt;br /&gt;
&lt;br /&gt;
== Parts ==&lt;br /&gt;
Broca's area contains two main parts: the ''Pars triangularis'' and the ''Pars opercularis''.&lt;br /&gt;
&lt;br /&gt;
The '''Pars triangularis''' is located in the anterior part of Broca's area. Researchers believe that this area of the brain is responsible for helping the human brain interpret different stimulus modes. It also supports the programming of verbal conducts.&lt;br /&gt;
&lt;br /&gt;
The '''Pars opercularis''' is located in the posterior part of Broca's area. It is believed that this area supports only one stimulus mode, rather than multiple modes like the Pars triangularis. This portion of Broca’s area is also believed to coordinate the organs used for speech in order to produce language.&lt;br /&gt;
&lt;br /&gt;
== Damage to Broca's area ==&lt;br /&gt;
If Broca's area is damaged, people will usually suffer from a condition called ''[[Broca's aphasia]]''. This condition is also sometimes called ''expressive aphasia'', ''nonfluent aphasia'', or ''motor aphasia''. Broca's aphasia makes people unable to create sentences that are grammatically complex. In addition, the sentences usually contain very few words related to content. Broca's aphasia is characterized by nonfluent speech, few words, short sentences, and many pauses.&lt;br /&gt;
&lt;br /&gt;
For example, if a Broca's aphasic was trying to explain how he came to the hospital for dental surgery, it might sound like this:&lt;br /&gt;
&lt;br /&gt;
&amp;quot;Yes... ah... Monday... er... Dad and Peter H... (his own name), and Dad.... er... hospital... and ah... Wednesday... Wednesday, nine o'clock... and oh... Thursday... ten o'clock, ah doctors... two... an' doctors... and er... teeth... yah.&amp;quot;&lt;br /&gt;
&lt;br /&gt;
Despite the Broca's aphasic's difficulty in putting together sentences, a person with a damaged Broca's area is generally capable of comprehending language without a problem. Sometimes, however, the person may have difficulty with understanding a few words used in a sentence with complex syntax. These people typically have damage only in the posterior part of Broca's area, a condition called ''[[Wernicke’s aphasia]]''. Those suffering from Wernicke’s aphasia may have somewhat normal speech, though it tends to be vague or even meaningless.&lt;br /&gt;
&lt;br /&gt;
== Video Clip ==&lt;br /&gt;
[http://www.youtube.com/watch?v=iuPeOKeCxu8 Broca's area video]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:03:52 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Broca%27s_area</comments>		</item>
		<item>
			<title>Broca's aphasia</title>
			<link>http://72.14.177.54/psy3241/Broca%27s_aphasia</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological syndromes]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Etiology ==&lt;br /&gt;
After a post-mortem autopsy of patient “[[Tan (aphasia patient)]]�? (nicknamed for the only sound he could successfully utter), [[Paul Broca]] discovered damage to the left posterior frontal gyrus (more specifically, the posterior surface of the third frontal gyrus).  This discovery, made in 1861, lead Broca to conclude that Tan’s lesion covered an area of the brain important for speech production.&lt;br /&gt;
&lt;br /&gt;
== Characteristics of Broca's Aphasia ==&lt;br /&gt;
Because the central feature of this aphasia involves difficulty producing coherent speech, alternative names include expressive or non-fluent aphasia.&lt;br /&gt;
“Telegraphic Speech�? is often used to categorize Broca’s aphasics, since their speech is deliberate, slow, and marked by several pauses.&lt;br /&gt;
The difficulties in their speech patterns often transfer into a comparable writing deficiency, suggesting that the problem is not related to physical muscle-movements associated with language production.&lt;br /&gt;
Prepositions, conjunctions, and other relational words are often omitted from speech, so Broca’s aphasics utilize a very simple grammatical structure, producing only the most basic of sentences. &lt;br /&gt;
Interestingly, personal catchphrases and emotional expressions are often stated quickly and clearly, which may point to processing in other areas of the brain for some language functions.&lt;br /&gt;
Generally speaking, Broca’s aphasics retain comprehension skills and have an understanding of their condition.  However, there is some evidence that Broca’s aphasics are impaired in their understanding of 3-step processes, complex language input, and numbers or symbols.&lt;br /&gt;
The lesion that causes Broca’s aphasia may also lead to a paralyzed right hand, paralysis of the right arm, right-sided sensory loss and, in rare cases, a visual-field defect. [http://www.youtube.com/watch?v=f2IiMEbMnPM Video of Broca's aphasia]&lt;br /&gt;
&lt;br /&gt;
== Relevant Articles/Case Presentations ==&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:02:30 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Broca%27s_aphasia</comments>		</item>
		<item>
			<title>Broca's aphasia</title>
			<link>http://72.14.177.54/psy3241/Broca%27s_aphasia</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Etiology */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological syndromes]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Etiology ==&lt;br /&gt;
After a post-mortem autopsy of patient “[[Tan (aphasia patient]]” (nicknamed for the only sound he could successfully utter), [[Paul Broca]] discovered damage to the left posterior frontal gyrus (more specifically, the posterior surface of the third frontal gyrus).  This discovery, made in 1861, lead Broca to conclude that Tan’s lesion covered an area of the brain important for speech production.&lt;br /&gt;
&lt;br /&gt;
== Characteristics of Broca's Aphasia ==&lt;br /&gt;
Because the central feature of this aphasia involves difficulty producing coherent speech, alternative names include expressive or non-fluent aphasia.&lt;br /&gt;
“Telegraphic Speech” is often used to categorize Broca’s aphasics, since their speech is deliberate, slow, and marked by several pauses.&lt;br /&gt;
The difficulties in their speech patterns often transfer into a comparable writing deficiency, suggesting that the problem is not related to physical muscle-movements associated with language production.&lt;br /&gt;
Prepositions, conjunctions, and other relational words are often omitted from speech, so Broca’s aphasics utilize a very simple grammatical structure, producing only the most basic of sentences. &lt;br /&gt;
Interestingly, personal catchphrases and emotional expressions are often stated quickly and clearly, which may point to processing in other areas of the brain for some language functions.&lt;br /&gt;
Generally speaking, Broca’s aphasics retain comprehension skills and have an understanding of their condition.  However, there is some evidence that Broca’s aphasics are impaired in their understanding of 3-step processes, complex language input, and numbers or symbols.&lt;br /&gt;
The lesion that causes Broca’s aphasia may also lead to a paralyzed right hand, paralysis of the right arm, right-sided sensory loss and, in rare cases, a visual-field defect. [http://www.youtube.com/watch?v=f2IiMEbMnPM Video of Broca's aphasia]&lt;br /&gt;
&lt;br /&gt;
== Relevant Articles/Case Presentations ==&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:01:53 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Broca%27s_aphasia</comments>		</item>
		<item>
			<title>Broca's aphasia</title>
			<link>http://72.14.177.54/psy3241/Broca%27s_aphasia</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Etiology */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological syndromes]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Etiology ==&lt;br /&gt;
After a post-mortem autopsy of patient “[[Tan]]” (nicknamed for the only sound he could successfully utter), [[Paul Broca]] discovered damage to the left posterior frontal gyrus (more specifically, the posterior surface of the third frontal gyrus).  This discovery, made in 1861, lead Broca to conclude that [[Tan]]’s lesion covered an area of the brain important for speech production.&lt;br /&gt;
&lt;br /&gt;
== Characteristics of Broca's Aphasia ==&lt;br /&gt;
Because the central feature of this aphasia involves difficulty producing coherent speech, alternative names include expressive or non-fluent aphasia.&lt;br /&gt;
“Telegraphic Speech” is often used to categorize Broca’s aphasics, since their speech is deliberate, slow, and marked by several pauses.&lt;br /&gt;
The difficulties in their speech patterns often transfer into a comparable writing deficiency, suggesting that the problem is not related to physical muscle-movements associated with language production.&lt;br /&gt;
Prepositions, conjunctions, and other relational words are often omitted from speech, so Broca’s aphasics utilize a very simple grammatical structure, producing only the most basic of sentences. &lt;br /&gt;
Interestingly, personal catchphrases and emotional expressions are often stated quickly and clearly, which may point to processing in other areas of the brain for some language functions.&lt;br /&gt;
Generally speaking, Broca’s aphasics retain comprehension skills and have an understanding of their condition.  However, there is some evidence that Broca’s aphasics are impaired in their understanding of 3-step processes, complex language input, and numbers or symbols.&lt;br /&gt;
The lesion that causes Broca’s aphasia may also lead to a paralyzed right hand, paralysis of the right arm, right-sided sensory loss and, in rare cases, a visual-field defect. [http://www.youtube.com/watch?v=f2IiMEbMnPM Video of Broca's aphasia]&lt;br /&gt;
&lt;br /&gt;
== Relevant Articles/Case Presentations ==&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 02:00:59 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Broca%27s_aphasia</comments>		</item>
		<item>
			<title>Broca's aphasia</title>
			<link>http://72.14.177.54/psy3241/Broca%27s_aphasia</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* Etiology */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological syndromes]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Etiology ==&lt;br /&gt;
After a post-mortem autopsy of patient “[[Tan]]” (nicknamed for the only sound he could successfully utter), Paul Broca discovered damage to the left posterior frontal gyrus (more specifically, the posterior surface of the third frontal gyrus).  This discovery, made in 1861, lead Broca to conclude that [[Tan]]’s lesion covered an area of the brain important for speech production.&lt;br /&gt;
&lt;br /&gt;
== Characteristics of Broca's Aphasia ==&lt;br /&gt;
Because the central feature of this aphasia involves difficulty producing coherent speech, alternative names include expressive or non-fluent aphasia.&lt;br /&gt;
“Telegraphic Speech” is often used to categorize Broca’s aphasics, since their speech is deliberate, slow, and marked by several pauses.&lt;br /&gt;
The difficulties in their speech patterns often transfer into a comparable writing deficiency, suggesting that the problem is not related to physical muscle-movements associated with language production.&lt;br /&gt;
Prepositions, conjunctions, and other relational words are often omitted from speech, so Broca’s aphasics utilize a very simple grammatical structure, producing only the most basic of sentences. &lt;br /&gt;
Interestingly, personal catchphrases and emotional expressions are often stated quickly and clearly, which may point to processing in other areas of the brain for some language functions.&lt;br /&gt;
Generally speaking, Broca’s aphasics retain comprehension skills and have an understanding of their condition.  However, there is some evidence that Broca’s aphasics are impaired in their understanding of 3-step processes, complex language input, and numbers or symbols.&lt;br /&gt;
The lesion that causes Broca’s aphasia may also lead to a paralyzed right hand, paralysis of the right arm, right-sided sensory loss and, in rare cases, a visual-field defect. [http://www.youtube.com/watch?v=f2IiMEbMnPM Video of Broca's aphasia]&lt;br /&gt;
&lt;br /&gt;
== Relevant Articles/Case Presentations ==&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 01:56:56 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Broca%27s_aphasia</comments>		</item>
		<item>
			<title>Wechsler adult intelligence scale</title>
			<link>http://72.14.177.54/psy3241/Wechsler_adult_intelligence_scale</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological methods]]&lt;br /&gt;
David Weschler created intelligence tests available to assess a wide range of ages. The first test, called the 'Weschler-Bellevue Intelligence Scale, was developed in 1939. It was replaced in 1955 and renamed the Weschler Adult Intelligence Scale. Again, another revision occurred in 1981 resulting in the Wechsler Adult Intelligence Scale-Revised. WAIS-R is the most standard assessment for measuring intellectual functioning (IQ). Its subtests include: Information, Picture completion, Digit Span, Picture arrangement, Vocabulary, Block design, Arithmetic, Object assembly, Comprehension, Digit symbol, and Similarities.&lt;br /&gt;
&lt;br /&gt;
A subsequent revision occurred in 1997 in the United States and only slightly revised it from its original form. This version is known as the WAIS-3rd edition or WAIS-III. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Johnstone, L., &amp;amp; Cooke, D. J. (2003). Feigned intellectual deficits on the Wechsler Adult Intelligence Scale-Revised. ''The British Journal of Clinical Psychology, 42''(3), 303-318.&lt;br /&gt;
&lt;br /&gt;
http://wilderdom.com/personality/intelligenceWAISWISC.html&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 01:49:29 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wechsler_adult_intelligence_scale</comments>		</item>
		<item>
			<title>Wechsler adult intelligence scale</title>
			<link>http://72.14.177.54/psy3241/Wechsler_adult_intelligence_scale</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological methods]]&lt;br /&gt;
David Weschler created intelligence tests available to assess a wide range of ages. The first test, called the 'Weschler-Bellevue Intelligence Scale, was developed in 1939. It was replaced in 1955 and renamed the Weschler Adult Intelligence Scale. Again, another revision occurred in 1981, which is now currently the Weschler Adult Intelligence Scale-Revised. &lt;br /&gt;
The Wechsler Adult Intelligence Scale-Revised (WAIS-R) is the most standard assessment for measuring intellectual functioning (IQ). Its subtests include: Information, Picture completion, Digit Span, Picture arrangement, Vocabulary, Block design, Arithmetic, Object assembly, Comprehension, Digit symbol, and Similarities.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Johnstone, L., &amp;amp; Cooke, D. J. (2003). Feigned intellectual deficits on the Wechsler Adult Intelligence Scale-Revised. ''The British Journal of Clinical Psychology, 42''(3), 303-318.&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 01:43:09 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wechsler_adult_intelligence_scale</comments>		</item>
		<item>
			<title>Alvaro Pascual-Leone</title>
			<link>http://72.14.177.54/psy3241/Alvaro_Pascual-Leone</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological profiles]]&lt;br /&gt;
[[Image:Images.jpg]]&lt;br /&gt;
&lt;br /&gt;
Alvaro Pascual-Leone is a neurophysiologist renowned for his work with Transcranial Magnetic Stimulation (TMS). Pascual-Leone and colleagues conducted a study published in 1995 observing the plasticity of motor cortical outputs to the reading hand in Braille readers. &lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Pascual-Leone, A., Wassermann, E. M., Sadato, N., &amp;amp; Hallett, M. (1995). The role of reading activity on the modulation of motor cortical outputs to the reading hand in Braille readers. ''Annals of Neurology, 38''(6), 910-915.&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 01:26:14 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Alvaro_Pascual-Leone</comments>		</item>
		<item>
			<title>Alvaro Pascual-Leone</title>
			<link>http://72.14.177.54/psy3241/Alvaro_Pascual-Leone</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological profiles]]&lt;br /&gt;
[[Image:Images.jpg]]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 01:14:27 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Alvaro_Pascual-Leone</comments>		</item>
		<item>
			<title>File:Images.jpg</title>
			<link>http://72.14.177.54/psy3241/File:Images.jpg</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 01:13:46 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/File_talk:Images.jpg</comments>		</item>
		<item>
			<title>Alexander Luria</title>
			<link>http://72.14.177.54/psy3241/Alexander_Luria</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological profiles]]&lt;br /&gt;
[[Image:bwl.jpg]]&lt;br /&gt;
&lt;br /&gt;
Alexander Romanovich Luria (1902-1977) is a Russian neurologist and psychologist. He has been cited by Solsa and colleagues as the most frequenly cited Russian psychology scholar in the North American continent. Luria proposed the brain as a functional system, in which different areas act together to result in behavior. &lt;br /&gt;
&lt;br /&gt;
Luria’s first book in the English language is &amp;quot;The Nature of Human Conflicts&amp;quot;. The book focuses on real emotions and the difficulty in studying them in a laboratory setting. First, he explains that emotions cannot be simulated by experimental manipulation and the result of this is artificial. Second, emotions should be studied in a real-life setting because it is important to examine the effects on voluntary behavior. To surmount the difficulties with real emotion, Luria developed combined motor method, an approach to map the effect emotions have on voluntary behavior. With this technique, Luria was able to develop real-life conflict models.&lt;br /&gt;
&lt;br /&gt;
His work in the neuropsychology field has been extremely influential; one of the more important topics in his work is his stance on localization and the cerebral organization of mental function. Tupper adds, &amp;quot;...Luria's neurolinguistic interests and aphasia classification, his drug and cognitive approaches to rehabilitation of individuals after brain injury, his discussions of breakdowns in regulatory activity associated with frontal lobe damage, and his qualitiative approach to assessment are all major areas of Luria's work&amp;quot; (Tupper 1999). In contemporary neuropsychology, Luria's theory of the three functional units of the brain is still an important learning method due to its simplicity.&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Tupper, D. E. (1999). Introduction: Alexander Luria's continuing influence on worldwide neuropsychology. ''Neuropsychology Review, 9''(1). 1-7.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
See also:  [[Luria-Nebraska battery]]&lt;/div&gt;</description>
			<pubDate>Tue, 29 Apr 2008 01:11:45 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Alexander_Luria</comments>		</item>
		<item>
			<title>Palmeri et al. (2002)</title>
			<link>http://72.14.177.54/psy3241/Palmeri_et_al._(2002)</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Synesthesia Symposium]]&lt;br /&gt;
&lt;br /&gt;
Introduction to WO &lt;br /&gt;
WO has lexical synesthesia, which means that achromatic words and alphanumeric characters are seen in bright and consistent colors. The subject, WO has experienced this form of synesthesia since childhood and describes that he experiences a visual imagery synesthesia in which spoken words evoke consistent colors in “his mind’s eye” (Pamleri et al., 2000, p.4127). Despite his synesthesia, he has normal trichromatic color vision, good stereopsis and normal visual acuity. &lt;br /&gt;
&lt;br /&gt;
Method and Results&lt;br /&gt;
The authors tested WO’s synesthesia as elicited by both local and global forms. (i.e. a large number “5” made of small “2s”) Depending on what the subject was attending to, the local or global form of the stimulus, WO experienced whatever color was consistently associated with that stimulus (i.e. light green for the “5” and orange for the “2s”). WO also elicited synesthesia in response to stereopic displays resulting from binocularly defined stimuli. The structure-from-motion defined stimuli, which consisted of arrows facing in different directions, which formed the percept of a number. WO readily demonstrated synesthesia by identifying the color for the number displayed. Palmeri et al. (2000) also conducted a modified stroop interference task in which WO was asked to name colors either congruent or incongruent with the ink color, using latency as the independent variable. WO showed significantly slower reaction time when the written color was incongruent with his synesthesia. In another modified stroop task WO was asked to name the color of the word, which was either congruent or incongruent with the colors normally evoked by his synesthetic experience. The reaction times evoked to name words printed in grey (control condition) had similar durations to the words printed in the appropriate synesthetic color. The final task included a visual search efficiency test consisting of comparable visual stimuli (i.e. a “2” among numerous “5s” or a “6” among numerous “8s”). The results showed that WO’s error rates and reaction times were significantly lower and than that of controls and he commented that the target number was easily distinguishable compared to the other digits. &lt;br /&gt;
&lt;br /&gt;
Discussion&lt;br /&gt;
&lt;br /&gt;
Synesthesia is an automatic condition that results in multimodal perceptual experiences from a unimodal sensory stimulus. Palmeri et. al. (2000) reported, “it appears that the binding of color and form takes place during the process of form recognition itself, with synesthetic colors available before the explicit recognition of the digits (p. 4130). The authors conclude that lexical synesthesia binds color to form before the synesthete consciously recognizes the stimulus. Thus, it seems that synesthesia occurs during central visual processing, instead during higher order, semantic processing.&lt;/div&gt;</description>
			<pubDate>Mon, 28 Apr 2008 19:08:37 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Palmeri_et_al._(2002)</comments>		</item>
		<item>
			<title>File:Bwl.jpg</title>
			<link>http://72.14.177.54/psy3241/File:Bwl.jpg</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;&lt;/div&gt;</description>
			<pubDate>Mon, 28 Apr 2008 00:46:48 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/File_talk:Bwl.jpg</comments>		</item>
		<item>
			<title>Alexander Luria</title>
			<link>http://72.14.177.54/psy3241/Alexander_Luria</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological profiles]]&lt;br /&gt;
&lt;br /&gt;
Alexander Romanovich Luria (1902-1977) is a Russian neurologist and psychologist. He has been cited by Solsa and colleagues as the most frequenly cited Russian psychology scholar in the North American continent. Luria proposed the brain as a functional system, in which different areas act together to result in behavior. &lt;br /&gt;
&lt;br /&gt;
Luria’s first book in the English language is &amp;quot;The Nature of Human Conflicts&amp;quot;. The book focuses on real emotions and the difficulty in studying them in a laboratory setting. First, he explains that emotions cannot be simulated by experimental manipulation and the result of this is artificial. Second, emotions should be studied in a real-life setting because it is important to examine the effects on voluntary behavior. To surmount the difficulties with real emotion, Luria developed combined motor method, an approach to map the effect emotions have on voluntary behavior. With this technique, Luria was able to develop real-life conflict models.&lt;br /&gt;
&lt;br /&gt;
His work in the neuropsychology field has been extremely influential; one of the more important topics in his work is his stance on localization and the cerebral organization of mental function. Tupper adds, &amp;quot;...Luria's neurolinguistic interests and aphasia classification, his drug and cognitive approaches to rehabilitation of individuals after brain injury, his discussions of breakdowns in regulatory activity associated with frontal lobe damage, and his qualitiative approach to assessment are all major areas of Luria's work&amp;quot; (Tupper 1999). In contemporary neuropsychology, Luria's theory of the three functional units of the brain is still an important learning method due to its simplicity.&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Tupper, D. E. (1999). Introduction: Alexander Luria's continuing influence on worldwide neuropsychology. ''Neuropsychology Review, 9''(1). 1-7.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
See also:  [[Luria-Nebraska battery]]&lt;/div&gt;</description>
			<pubDate>Mon, 28 Apr 2008 00:35:49 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Alexander_Luria</comments>		</item>
		<item>
			<title>Alexander Luria</title>
			<link>http://72.14.177.54/psy3241/Alexander_Luria</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological profiles]]&lt;br /&gt;
&lt;br /&gt;
Alexander Romanovich Luria (1902-1977) is a Russian neurologist and psychologist. He has been cited by Solsa and colleagues as the most frequenly cited Russian psychology scholar in the North American continent. Luria proposed the brain as a functional system, in which different areas act together to result in behavior. &lt;br /&gt;
Luria’s first book in the English language is The Nature of Human Conflicts. The book focuses on real emotions and the difficulty in studying them in a laboratory setting. First, he explains that emotions cannot be simulated by experimental manipulation and the result of this is artificial. Second, emotions should be studied in a real-life setting because it is important to examine the effects on voluntary behavior. To surmount the difficulties with real emotion, Luria developed combined motor method, an approach to map the effect emotions have on voluntary behavior. With this technique, Luria was able to develop real-life conflict models.&lt;br /&gt;
His work in the neuropsychology field has been extremely influential; one of the more important topics in his work is his stance on localization and the cerebral organization of mental function. Tupper adds, &amp;quot;...Luria's neurolinguistic interests and aphasia classification, his drug and cognitive approaches to rehabilitation of individuals after brain injury, his discussions of breakdowns in regulatory activity associated with frontal lobe damage, and his qualitiative approach to assessment are all major areas of Luria's work&amp;quot; (Tupper 1999). In contemporary neuropsychology, Luria's theory of the three functional units of the brain is still an important learning method due to its simplicity.&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Tupper, D. E. (1999). Introduction: Alexander Luria's continuing influence on worldwide neuropsychology. ''Neuropsychology Review, 9''(1). 1-7.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
See also:  [[Luria-Nebraska battery]]&lt;/div&gt;</description>
			<pubDate>Mon, 28 Apr 2008 00:29:18 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Alexander_Luria</comments>		</item>
		<item>
			<title>Alexander Luria</title>
			<link>http://72.14.177.54/psy3241/Alexander_Luria</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological profiles]]&lt;br /&gt;
&lt;br /&gt;
Alexander Romanovich Luria (1902-1977) is a Russian neurologist and psychologist. He has been cited by Solsa and colleagues as the most frequenly cited Russian psychology scholar in the North American continent. Luria proposed the brain as a functional system, in which different areas acting together to result in behavior. His work in the neuropsychology field has been extremely influential; one of the more important topics in his work is his stance on localization and the cerebral organization of mental function. Tupper adds, &amp;quot;...Luria's neurolinguistic interests and aphasia classification, his drug and cognitive approaches to rehabilitation of individuals after brain injury, his discussions of breakdowns in regulatory activity associated with frontal lobe damage, and his qualitiative approach to assessment are all major areas of Luria's work&amp;quot; (Tupper 1999).&lt;/div&gt;</description>
			<pubDate>Sun, 27 Apr 2008 23:04:56 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Alexander_Luria</comments>		</item>
		<item>
			<title>Alexander Luria</title>
			<link>http://72.14.177.54/psy3241/Alexander_Luria</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological profiles]]&lt;br /&gt;
&lt;br /&gt;
Alexander Romanovich Luria (1902-1977) is a Russian neurologist and psychologist. He has been cited by Solsa and colleagues as the most frequenly cited Russian psychology scholar in the North American continent. Luria proposed the brain as a functional system, in which different areas acting together to result in behavior.&lt;/div&gt;</description>
			<pubDate>Thu, 24 Apr 2008 20:09:29 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Alexander_Luria</comments>		</item>
		<item>
			<title>Amnesia</title>
			<link>http://72.14.177.54/psy3241/Amnesia</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* ECT-Induced Amnesia */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological syndromes]]&lt;br /&gt;
Amnesia is a memory deficit, resulting from brain damage, disease, or injury. Memory loss may be selective or generalized, temporary or permanent, and it may affect short-term memory, long-term memory, or both.&lt;br /&gt;
Research for amnesia is does not have the ability to be experimentally manipulated. Therefore, amnesia information is reliant on amnesic individuals who volunteer for research.&lt;br /&gt;
&lt;br /&gt;
== Diencephalic Amnesia ==&lt;br /&gt;
Damage to the diencephalic structues also has the ability to lead to memory impairments. Cases of diencephalic amnesia were used in a study by Knowlton et. al. hypothesizing separate but parallel learning systems. In patients with amnesia, either with damage to the hippocampal formation or diencephalic midline, participants performed normally on a probabilistic classification task. However, scores for a declarative memory task were poor. The results were opposite for participants with Parkinson's disease. These results demonstrate declarative memory is dependent on the medial temporal lobe or diencephalon, but not on the neostriatum. The opposite is the case for probabilistic classification learning.&lt;br /&gt;
&lt;br /&gt;
A 'pure' case of diencephalic amnesia comes in the form of NA. After suffering a freak accident involving a fencing foil, NA had damage to his left dorsal thalamus, his mamillary bodies (bi-laterally), and his mamillo-thalamic tract. He showed normal short-term memory, but was severely impaired in declarative long-term memory, particularly for verbal material. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Global Amnesia ==&lt;br /&gt;
Global amnesia is usually a result of bilateral damage to structures on the internal aspects of the cerebral hemispheres. It is characterized by an inability to learn/retain new information, including verbal, nonverbal, visual, or auditory, as well as a period of retrograde amnesia. Those suffering from Korsakoff's disease as well as patients who survive herpes simplex encephalitis may demonstrate global amnesia.&lt;br /&gt;
&lt;br /&gt;
== Frontal Amnesia ==&lt;br /&gt;
Frontal amnesia is characterized as memory deficits resulting from frontal lobe damage. Typically, the patient is impaired in learning and recalling new information.&lt;br /&gt;
&lt;br /&gt;
== Korsakoff's Syndrome ==&lt;br /&gt;
[[http://editthis.info/psy3241/Korsakoff%27s_syndrome]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Concussion Amnesia ==&lt;br /&gt;
Concussion-induced amnesia almost always results in ''temporary'' memory impairment, unless there was organic damage to the brain. A typical case may include both anterograde (after on-set) amnesia and retrograde (before accident) amnesia. A unique feature of concussion amnesia is that memory loss usually is partially recovered with time, although there is nearly always some permanent loss. There may be impairments in the consolidation of new information from short-term to long-term memory. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== ECT-Induced Amnesia ==&lt;br /&gt;
Electroconvulsive therapy involves the induction of epileptic seizures by electric current. ECT-induced amnesia resembles that of concussion amnesia. There is both anterograde and retrograde amnesia, but also shrinkage over time. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
Knowlton, B. J., Mangels, J. A., &amp;amp; Squire, L. R. (1996). A neostriatal habit learning system in humans. ''Science, 273''(5280), 1399-1402.&lt;br /&gt;
&lt;br /&gt;
Stirling, J. (2002). ''Introducing neuropsychology.'' New York: Psychology Press.&lt;br /&gt;
&lt;br /&gt;
Ogden, J. A. (2005). ''Fractured minds.'' New York: Oxford University Press.&lt;br /&gt;
&lt;br /&gt;
== External Links ==&lt;br /&gt;
[http://www.youtube.com/watch?v=5ObnErfTblY Amnesia]&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:55:48 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Amnesia</comments>		</item>
		<item>
			<title>Wechsler adult intelligence scale</title>
			<link>http://72.14.177.54/psy3241/Wechsler_adult_intelligence_scale</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological methods]]&lt;br /&gt;
The Wechsler Adult Intelligence Scale-Revised (WAIS-R) is the most standard assessment for measuring intellectual functioning (IQ). Its subtests include: Information, Picture completion, Digit Span, Picture arrangement, Vocabulary, Block design, Arithmetic, Object assembly, Comprehension, Digit symbol, and Similarities.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Johnstone, L., &amp;amp; Cooke, D. J. (2003). Feigned intellectual deficits on the Wechsler Adult Intelligence Scale-Revised. ''The British Journal of Clinical Psychology, 42''(3), 303-318.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:53:53 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wechsler_adult_intelligence_scale</comments>		</item>
		<item>
			<title>Wechsler adult intelligence scale</title>
			<link>http://72.14.177.54/psy3241/Wechsler_adult_intelligence_scale</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological methods]]&lt;br /&gt;
The Wechsler Adult Intelligence Scale-Revised (WAIS-R) is the most standard assessment for measuring intellectual functioning. Its subtests include: Information, Picture completion, Digit Span, Picture arrangement, Vocabulary, Block design, Arithmetic, Object assembly, Comprehension, Digit symbol, and Similarities.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:40:46 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wechsler_adult_intelligence_scale</comments>		</item>
		<item>
			<title>Wechsler adult intelligence scale</title>
			<link>http://72.14.177.54/psy3241/Wechsler_adult_intelligence_scale</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological methods]]&lt;br /&gt;
The Wechsler Adult Intelligence Scale-Revised (WAIS-R) is the most standard assessment for measuring intellectual functioning. Its subtests include: Information, Picture completion, Digit Span (F), Digit Span (B), Picture arrangement, Vocabulary, Block design, Arithmetic, Object assembly, Comprehension, Digit symbol, and Similarities.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:40:15 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wechsler_adult_intelligence_scale</comments>		</item>
		<item>
			<title>Alexander Luria</title>
			<link>http://72.14.177.54/psy3241/Alexander_Luria</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological profiles]]&lt;br /&gt;
&lt;br /&gt;
Alexander Romanovich Luria (1902-1977) is a Russian neurologist and psychologist. He has been cited by Solsa and colleagues as the most frequenly cited Russian psychology scholar in the North American continent.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:10:47 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Alexander_Luria</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[Image:wernickesarea.gif]]&lt;br /&gt;
&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. Specifically, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to Broca's area. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. Recent research by Dronkers et. al. (1998) revealed that 'pure' damage to Wernicke's area most likely results in impairment in repetition rather than comprehension deficits. Damage to the part of the brain linking Broca's area and Wernicke's area, the arcuate fasciculus, can lead to conduction aphasia, in which the patient loses the ability to repeat words.&lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
Stirling, J. (2002). Introducing neuropsychology. New York: Psychology Press.&lt;br /&gt;
&lt;br /&gt;
Ogden, J. A. (2005). Fractured minds. New York: Oxford University Press. &lt;br /&gt;
&lt;br /&gt;
Image taken from: &lt;br /&gt;
http://users.fmrib.ox.ac.uk/~stuart/thesis/chapter_3/section3_2.html&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:08:56 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* References */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[Image:wernickesarea.gif]]&lt;br /&gt;
&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. Specifically, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to Broca's area. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. Recent research by Dronkers et. al. (1998) revealed that 'pure' damage to Wernicke's area most likely results in impairment in repetition rather than comprehension deficits. Damage to the part of the brain linking Broca's area and Wernicke's area, the arcuate fasciculus, can lead to conduction aphasia, in which the patient loses the ability to repeat words.&lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
Image taken from: &lt;br /&gt;
http://users.fmrib.ox.ac.uk/~stuart/thesis/chapter_3/section3_2.html&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:07:57 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* References */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[Image:wernickesarea.gif]]&lt;br /&gt;
&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. Specifically, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to Broca's area. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. Recent research by Dronkers et. al. (1998) revealed that 'pure' damage to Wernicke's area most likely results in impairment in repetition rather than comprehension deficits. Damage to the part of the brain linking Broca's area and Wernicke's area, the arcuate fasciculus, can lead to conduction aphasia, in which the patient loses the ability to repeat words.&lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
Image taken from: &lt;br /&gt;
http://users.fmrib.ox.ac.uk/~stuart/thesis/chapter_3/image3_15.gif&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:07:21 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[Image:wernickesarea.gif]]&lt;br /&gt;
&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. Specifically, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to Broca's area. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. Recent research by Dronkers et. al. (1998) revealed that 'pure' damage to Wernicke's area most likely results in impairment in repetition rather than comprehension deficits. Damage to the part of the brain linking Broca's area and Wernicke's area, the arcuate fasciculus, can lead to conduction aphasia, in which the patient loses the ability to repeat words.&lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
Image taken from: http://images.google.com/imgres?imgurl=http://users.fmrib.ox.ac.uk/~stuart/thesis/chapter_3/image3_15.gif&amp;amp;imgrefurl=http://users.fmrib.ox.ac.uk/~stuart/thesis/chapter_3/section3_2.html&amp;amp;h=433&amp;amp;w=394&amp;amp;sz=46&amp;amp;hl=en&amp;amp;start=6&amp;amp;um=1&amp;amp;tbnid=1SES40aaFRGVSM:&amp;amp;tbnh=126&amp;amp;tbnw=115&amp;amp;prev=/images%3Fq%3Dwernicke%2527s%2Barea%26um%3D1%26hl%3Den%26client%3Dfirefox-a%26rls%3Dorg.mozilla:en-US:official%26sa%3DN&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:06:21 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[Image:wernickesarea.gif]]&lt;br /&gt;
&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. Specifically, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to Broca's area. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. Damage to the part of the brain linking Broca's area and Wernicke's area, arcuate fasciculus, can lead to conduction aphasia, in which the patient loses the ability to repeat words.&lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 17:00:04 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[Image:wernickesarea.gif]]&lt;br /&gt;
&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. Specifically, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to Broca's area. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. &lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 16:57:41 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>File:Wernickesarea.gif</title>
			<link>http://72.14.177.54/psy3241/File:Wernickesarea.gif</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 16:57:00 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/File_talk:Wernickesarea.gif</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[Image:wernickesarea.gif]]&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. Specifically, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to Broca's area. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. &lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 16:56:11 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
[[wernickesarea.gif]]&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. Specifically, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to Broca's area. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. &lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 16:55:48 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. Specifically, this area controls the function of connects speech sounds to stored representations of words. Wernicke's area is anatomically linked to Broca's area. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. &lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 16:52:29 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Brain areas]]&lt;br /&gt;
Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus. A lesion to this area will likely result in difficulties in language comprehension. In an article published by Carl Wernicke in 1874, he reported 10 aphasic patients with difficulties in language comprehension. An autopsy on four of the patients provided results that they had lesions damaging the left temporal lobe. This specfic type of aphasia is now known as ''Wernicke's aphasia''. &lt;br /&gt;
&lt;br /&gt;
Other brain areas associated with language function include: Broca's area, supramarginal gyrus, angular gyrus, and arcuate fasciculus.&lt;/div&gt;</description>
			<pubDate>Wed, 23 Apr 2008 16:43:23 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Wernicke's area</title>
			<link>http://72.14.177.54/psy3241/Wernicke%27s_area</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;Wernicke's area was discovered by Karl Wernicke in 1874. It is the left superior temporal gyrus.&lt;/div&gt;</description>
			<pubDate>Tue, 22 Apr 2008 23:50:35 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Wernicke%27s_area</comments>		</item>
		<item>
			<title>Alexander Luria</title>
			<link>http://72.14.177.54/psy3241/Alexander_Luria</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;Alexander Romanovich Luria (1902-1977) is a Russian neurologist and psychologist. He has been cited by Solsa and colleagues as the most frequenly cited Russian psychology scholar in the North American continent.&lt;/div&gt;</description>
			<pubDate>Tue, 22 Apr 2008 23:31:34 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Alexander_Luria</comments>		</item>
		<item>
			<title>Amnesia</title>
			<link>http://72.14.177.54/psy3241/Amnesia</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* References */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological syndromes]]&lt;br /&gt;
Amnesia is a memory deficit, resulting from brain damage, disease, or injury. Memory loss may be selective or generalized, temporary or permanent, and it may affect short-term memory, long-term memory, or both.&lt;br /&gt;
Research for amnesia is does not have the ability to be experimentally manipulated. Therefore, amnesia information is reliant on amnesic individuals who volunteer for research.&lt;br /&gt;
&lt;br /&gt;
== Diencephalic Amnesia ==&lt;br /&gt;
Damage to the diencephalic structues also has the ability to lead to memory impairments. Cases of diencephalic amnesia were used in a study by Knowlton et. al. hypothesizing separate but parallel learning systems. In patients with amnesia, either with damage to the hippocampal formation or diencephalic midline, participants performed normally on a probabilistic classification task. However, scores for a declarative memory task were poor. The results were opposite for participants with Parkinson's disease. These results demonstrate declarative memory is dependent on the medial temporal lobe or diencephalon, but not on the neostriatum. The opposite is the case for probabilistic classification learning.&lt;br /&gt;
&lt;br /&gt;
A 'pure' case of diencephalic amnesia comes in the form of NA. After suffering a freak accident involving a fencing foil, NA had damage to his left dorsal thalamus, his mamillary bodies (bi-laterally), and his mamillo-thalamic tract. He showed normal short-term memory, but was severely impaired in declarative long-term memory, particularly for verbal material. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Global Amnesia ==&lt;br /&gt;
Global amnesia is usually a result of bilateral damage to structures on the internal aspects of the cerebral hemispheres. It is characterized by an inability to learn/retain new information, including verbal, nonverbal, visual, or auditory, as well as a period of retrograde amnesia. Those suffering from Korsakoff's disease as well as patients who survive herpes simplex encephalitis may demonstrate global amnesia.&lt;br /&gt;
&lt;br /&gt;
== Frontal Amnesia ==&lt;br /&gt;
Frontal amnesia is characterized as memory deficits resulting from frontal lobe damage. Typically, the patient is impaired in learning and recalling new information.&lt;br /&gt;
&lt;br /&gt;
== Korsakoff's Syndrome ==&lt;br /&gt;
[[http://editthis.info/psy3241/Korsakoff%27s_syndrome]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Concussion Amnesia ==&lt;br /&gt;
Concussion-induced amnesia almost always results in ''temporary'' memory impairment, unless there was organic damage to the brain. A typical case may include both anterograde (after on-set) amnesia and retrograde (before accident) amnesia. A unique feature of concussion amnesia is that memory loss usually is partially recovered with time, although there is nearly always some permanent loss. There may be impairments in the consolidation of new information from short-term to long-term memory. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== ECT-Induced Amnesia ==&lt;br /&gt;
Electroconvulsive therapy involves the induction of epileptic seizures by electric current. ECT-induced amnesia resembles that of concussion amnesia. There is both anterograde and retrograde amnesia, but also shrinkage over time. &lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
Knowlton, B. J., Mangels, J. A., &amp;amp; Squire, L. R. (1996). A neostriatal habit learning system in humans. ''Science, 273''(5280), 1399-1402.&lt;br /&gt;
&lt;br /&gt;
Stirling, J. (2002). ''Introducing neuropsychology.'' New York: Psychology Press.&lt;br /&gt;
&lt;br /&gt;
Ogden, J. A. (2005). ''Fractured minds.'' New York: Oxford University Press.&lt;/div&gt;</description>
			<pubDate>Tue, 22 Apr 2008 04:41:34 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Amnesia</comments>		</item>
		<item>
			<title>Amnesia</title>
			<link>http://72.14.177.54/psy3241/Amnesia</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* References */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological syndromes]]&lt;br /&gt;
Amnesia is a memory deficit, resulting from brain damage, disease, or injury. Memory loss may be selective or generalized, temporary or permanent, and it may affect short-term memory, long-term memory, or both.&lt;br /&gt;
Research for amnesia is does not have the ability to be experimentally manipulated. Therefore, amnesia information is reliant on amnesic individuals who volunteer for research.&lt;br /&gt;
&lt;br /&gt;
== Diencephalic Amnesia ==&lt;br /&gt;
Damage to the diencephalic structues also has the ability to lead to memory impairments. Cases of diencephalic amnesia were used in a study by Knowlton et. al. hypothesizing separate but parallel learning systems. In patients with amnesia, either with damage to the hippocampal formation or diencephalic midline, participants performed normally on a probabilistic classification task. However, scores for a declarative memory task were poor. The results were opposite for participants with Parkinson's disease. These results demonstrate declarative memory is dependent on the medial temporal lobe or diencephalon, but not on the neostriatum. The opposite is the case for probabilistic classification learning.&lt;br /&gt;
&lt;br /&gt;
A 'pure' case of diencephalic amnesia comes in the form of NA. After suffering a freak accident involving a fencing foil, NA had damage to his left dorsal thalamus, his mamillary bodies (bi-laterally), and his mamillo-thalamic tract. He showed normal short-term memory, but was severely impaired in declarative long-term memory, particularly for verbal material. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Global Amnesia ==&lt;br /&gt;
Global amnesia is usually a result of bilateral damage to structures on the internal aspects of the cerebral hemispheres. It is characterized by an inability to learn/retain new information, including verbal, nonverbal, visual, or auditory, as well as a period of retrograde amnesia. Those suffering from Korsakoff's disease as well as patients who survive herpes simplex encephalitis may demonstrate global amnesia.&lt;br /&gt;
&lt;br /&gt;
== Frontal Amnesia ==&lt;br /&gt;
Frontal amnesia is characterized as memory deficits resulting from frontal lobe damage. Typically, the patient is impaired in learning and recalling new information.&lt;br /&gt;
&lt;br /&gt;
== Korsakoff's Syndrome ==&lt;br /&gt;
[[http://editthis.info/psy3241/Korsakoff%27s_syndrome]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Concussion Amnesia ==&lt;br /&gt;
Concussion-induced amnesia almost always results in ''temporary'' memory impairment, unless there was organic damage to the brain. A typical case may include both anterograde (after on-set) amnesia and retrograde (before accident) amnesia. A unique feature of concussion amnesia is that memory loss usually is partially recovered with time, although there is nearly always some permanent loss. There may be impairments in the consolidation of new information from short-term to long-term memory. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== ECT-Induced Amnesia ==&lt;br /&gt;
Electroconvulsive therapy involves the induction of epileptic seizures by electric current. ECT-induced amnesia resembles that of concussion amnesia. There is both anterograde and retrograde amnesia, but also shrinkage over time. &lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
Knowlton, B. J., Mangels, J. A., &amp;amp; Squire, L. R. (1996). A neostriatal habit learning system in humans. ''Science, 273''(5280), 1399-1402.&lt;br /&gt;
Stirling, J. (2002). ''Introducing neuropsychology.'' New York: Psychology Press.&lt;br /&gt;
&lt;br /&gt;
Ogden, J. A. (2005). ''Fractured minds.'' New York: Oxford University Press.&lt;/div&gt;</description>
			<pubDate>Tue, 22 Apr 2008 04:41:25 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Amnesia</comments>		</item>
		<item>
			<title>Amnesia</title>
			<link>http://72.14.177.54/psy3241/Amnesia</link>
			<description>&lt;p&gt;Kgutekunst:&amp;#32;/* References */&lt;/p&gt;
&lt;hr /&gt;
&lt;div&gt;[[Category:Neuropsychological syndromes]]&lt;br /&gt;
Amnesia is a memory deficit, resulting from brain damage, disease, or injury. Memory loss may be selective or generalized, temporary or permanent, and it may affect short-term memory, long-term memory, or both.&lt;br /&gt;
Research for amnesia is does not have the ability to be experimentally manipulated. Therefore, amnesia information is reliant on amnesic individuals who volunteer for research.&lt;br /&gt;
&lt;br /&gt;
== Diencephalic Amnesia ==&lt;br /&gt;
Damage to the diencephalic structues also has the ability to lead to memory impairments. Cases of diencephalic amnesia were used in a study by Knowlton et. al. hypothesizing separate but parallel learning systems. In patients with amnesia, either with damage to the hippocampal formation or diencephalic midline, participants performed normally on a probabilistic classification task. However, scores for a declarative memory task were poor. The results were opposite for participants with Parkinson's disease. These results demonstrate declarative memory is dependent on the medial temporal lobe or diencephalon, but not on the neostriatum. The opposite is the case for probabilistic classification learning.&lt;br /&gt;
&lt;br /&gt;
A 'pure' case of diencephalic amnesia comes in the form of NA. After suffering a freak accident involving a fencing foil, NA had damage to his left dorsal thalamus, his mamillary bodies (bi-laterally), and his mamillo-thalamic tract. He showed normal short-term memory, but was severely impaired in declarative long-term memory, particularly for verbal material. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Global Amnesia ==&lt;br /&gt;
Global amnesia is usually a result of bilateral damage to structures on the internal aspects of the cerebral hemispheres. It is characterized by an inability to learn/retain new information, including verbal, nonverbal, visual, or auditory, as well as a period of retrograde amnesia. Those suffering from Korsakoff's disease as well as patients who survive herpes simplex encephalitis may demonstrate global amnesia.&lt;br /&gt;
&lt;br /&gt;
== Frontal Amnesia ==&lt;br /&gt;
Frontal amnesia is characterized as memory deficits resulting from frontal lobe damage. Typically, the patient is impaired in learning and recalling new information.&lt;br /&gt;
&lt;br /&gt;
== Korsakoff's Syndrome ==&lt;br /&gt;
[[http://editthis.info/psy3241/Korsakoff%27s_syndrome]]&lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== Concussion Amnesia ==&lt;br /&gt;
Concussion-induced amnesia almost always results in ''temporary'' memory impairment, unless there was organic damage to the brain. A typical case may include both anterograde (after on-set) amnesia and retrograde (before accident) amnesia. A unique feature of concussion amnesia is that memory loss usually is partially recovered with time, although there is nearly always some permanent loss. There may be impairments in the consolidation of new information from short-term to long-term memory. &lt;br /&gt;
&lt;br /&gt;
&lt;br /&gt;
== ECT-Induced Amnesia ==&lt;br /&gt;
Electroconvulsive therapy involves the induction of epileptic seizures by electric current. ECT-induced amnesia resembles that of concussion amnesia. There is both anterograde and retrograde amnesia, but also shrinkage over time. &lt;br /&gt;
&lt;br /&gt;
== References ==&lt;br /&gt;
Knowlton, B. J., Mangels, J. A., &amp;amp; Squire, L. R. (1996). A neostriatal habit learning system in humans. ''Science, 273''(5280), 1399-1402.&lt;br /&gt;
Stirling, J. (2002). ''Introducing neuropsychology.'' New York: Psychology Press.&lt;br /&gt;
Ogden, J. A. (2005). ''Fractured minds.'' New York: Oxford University Press.&lt;/div&gt;</description>
			<pubDate>Tue, 22 Apr 2008 04:40:45 GMT</pubDate>			<dc:creator>Kgutekunst</dc:creator>			<comments>http://72.14.177.54/psy3241/Talk:Amnesia</comments>		</item>
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